Neonatal exposure to monosodium glutamate induces cell death and dendritic hypotrophy in rat prefrontocortical pyramidal neurons
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Acknowledgements
This work was partially supported by CONACyT, under reference number 90301-M, to CBZ.
References (20)
- et al.
Locomotor and learning deficits in adult rats exposed to monosodium-l-glutamate during early life
Neurosci. Lett.
(2000) - et al.
Excitotoxicity in neurological disorders – the glutamate paradox
Int. J. Dev. Neurosci.
(2000) - et al.
Prenatal and postnatal exposure to ethanol induces changes in the shape of the dendritic spines from hippocampal CA1 pyramidal neurons of the rat
Neurosci. Lett.
(2000) Calcium- and activity-dependent synaptic plasticity
Curr. Opin. Neurobiol.
(1999)- et al.
Effect of neonatal monosodium glutamate on the activities of glutamate dehydrogenase and aminotransferases in the circumventricular organs of the rat brain
Amino Acids
(1994) - et al.
Effect of neonatal monosodium glutamate on lipid peroxidation in adult rat brain
NeuroReport
(1995) - et al.
Ontogeny of non-NMDA glutamate receptors in rat barrel field cortex: α-AMPA and kainate receptors
J. Comp. Neurol.
(1997) - et al.
A role for N-methyl-d-aspartate receptors in the regulation of synaptogenesis and expression of the polysialylated form of the neural cell adhesion molecule in the developing striatum
Dev. Neurosci.
(1998) - et al.
Optima preservación ultraestructural del sistema nervioso central por perfusión intravascular de glutaraldehı́do
Arch. Inv. Med.
(1970) - et al.
Laminar segregation of the cortical plate during corticogenesis is accompanied by changes in glutamate receptor expression
Neurobiology
(1999)
There are more references available in the full text version of this article.
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