Abstract
Both the observed cis-inhibition and the proposed trans-activation of the insulin receptor tyrosine kinase help explain insulin signalling through its receptor.
MeSH terms
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Amino Acid Sequence
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Animals
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Binding Sites
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Humans
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Insulin / physiology*
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Models, Molecular
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Protein Conformation*
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Protein Structure, Secondary
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Receptor, Insulin / biosynthesis
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Receptor, Insulin / chemistry*
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Receptor, Insulin / physiology
Substances
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Insulin
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Receptor, Insulin